HMGB1/2 acted as a target for corynoxine B (Cory B) to promote

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Fe65-engineered neuronal exosomes encapsulating corynoxine-B ameliorate cognition and pathology of Alzheimer's disease

HMGB1 is involved in autophagy inhibition caused by SNCA/α-synuclein overexpression: a process modulated by the natural autophagy inducer corynoxine B. - Abstract - Europe PMC

Juxian SONG, Research Assistant Professor, PhD, Hong Kong Baptist University, Kowloon, School of Chinese Medicine

Qi Zhu's research works University of Macau, Macau and other places

High‐mobility group box 1 in Parkinson's disease: from pathogenesis to therapeutic approaches - Angelopoulou - 2018 - Journal of Neurochemistry - Wiley Online Library

Juxian SONG, Research Assistant Professor, PhD, Hong Kong Baptist University, Kowloon, School of Chinese Medicine

Corynoxine B targets at HMGB1/2 to enhance autophagy for α-synuclein clearance in fly and rodent models of Parkinson's disease - ScienceDirect

Biomolecules, Free Full-Text

Fe65-engineered neuronal exosomes encapsulating corynoxine-B ameliorate cognition and pathology of Alzheimer's disease

Loss of HMGB1 exacerbates DSS and Il10 –/– colitis. ( A ) Survival